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IVERMECTIN

William Briggs:

 

IVERMECTIN

New paper: Role of ivermectin in the prevention of COVID-19 infection among healthcare workers in India: A matched case-control study.

The Abstract:

A hospital-based matched case-control study was conducted among healthcare workers of AIIMS Bhubaneswar, India, from September to October 2020. Profession, gender, age and date of diagnosis were matched for 186 case-control pairs…

Results Ivermectin prophylaxis was taken by 77 controls and 38 cases. Two-dose ivermectin prophylaxis (0.27, 95% CI, 0.15-0.51) was associated with 73% reduction of COVID-19 infection among healthcare workers for the following one month, those who were involved in physical activity (3.06 95% CI, 1.18-7.93) for more than an hour/day were more likely to contract COVID-19 infection. Type of household, COVID duty, single-dose ivermectin prophylaxis, vitamin-C prophylaxis and hydroxychloroquine prophylaxis were not associated with COVID-19 infection.

Conclusion Two-dose ivermectin prophylaxis at a dose of 300 ?g/kg with a gap of 72 hours was associated 73% reduction of COVID-19 infection among healthcare workers for the following one-month.

Item: Latest peer-reviewed research: Immediate global ivermectin use will end COVID-19 pandemic.

o evaluate the efficacy of ivermectin in preventing COVID-19, 3 RCT’s and 5 observational controlled trial’s including almost 2,500 patients all reported that ivermectin significantly reduces the risk of contracting COVID-19 when used regularly.

Don’t share these on social media. Evidence counter to regime narratives is denial of The Science.

Even if you’ve won a Nobel Prize in Medicine. Which is nothing next to the knowledge of blue-haired landwhale censors at YouTube. (Thanks to John Cook for the tip.)

VEXXINES

Old paper: Vaccine-induced enhancement of viral infections.

The Abstract:

Examples of vaccine-induced enhancement of susceptibility to virus infection or of aberrant viral pathogenesis have been documented for infections by members of different virus families. Several mechanisms, many of which still are poorly understood, are at the basis of this phenomenon. Vaccine development for lentivirus infections in general, and for HIV/AIDS in particular, has been little successful. Certain experimental lentiviral vaccines even proved to be counterproductive: they rendered vaccinated subjects more susceptible to infection rather than protecting them. For vaccine-induced enhanced susceptibility to infection with certain viruses like feline coronavirus, Dengue virus, and feline immunodeficiency virus, it has been shown that antibody-dependent enhancement (ADE) plays an important role. Other mechanisms may, either in the absence of or in combination with ADE, be involved. Consequently, vaccine-induced enhancement has been a major stumble block in the development of certain flavi-, corona-, paramyxo-, and lentivirus vaccines. Also recent failures in the development of a vaccine against HIV may at least in part be attributed to induction of enhanced susceptibility to infection. There may well be a delicate balance between the induction of protective immunity on the one hand and the induction of enhanced susceptibility on the other. The present paper reviews the currently known mechanisms of vaccine-induced enhancement of susceptibility to virus infection or of aberrant viral pathogenesis.

Don’t worry. We know this won’t happen with the experimental coronadoom vexxines. We know because Experts forbid us to discuss it. [https://wmbriggs.com/post/36450/]

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